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Subject: Genital Wart Treatment
Description: genital wart treatment for condyloma caused by sexually transmitted disease hpv virus
Category: Sexually Transmitted Diseases

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Genital Warts

More than 30 types of HPV can infect the genital tract.

Most HPV infections are asymptomatic, unrecognized, or subclinical.

Visible genital warts usually are caused by HPV types 6 or or 11.

Other HPV types in stds the anogenital region (e.g., types 16, 18, 31, 33, and 35) have been strongly associated with cervical neoplasia.

Diagnosis of genital warts can be confirmed by biopsy, although although biopsy is needed only under certain circumstances (e.g., if stds the diagnosis is uncertain; the lesions do not respond to standard therapy; the disease worsens during therapy; the patient is immunocompromised; or warts are stds are pigmented, indurated, fixed, and ulcerated) .

No data support the use of type-specific HPV nucleic acid tests in the routine diagnosis or management of visible genital warts.

In addition to the external external genitalia (i.e., stds the penis, vulva, scrotum, perineum, and perianal skin) , genital warts can occur on the uterine cervix and in the vagina, urethra, anus, and mouth; these warts are sometimes symptomatic.

Intra-anal Intra-anal warts stds are seen predominantly in patients who have had receptive anal intercourse; these warts are distinct from perianal warts, which can occur in men and women who do not have a history of of anal sex.

In addition to stds the genital area, HPV types 6 and 11 have been associated with conjunctival, nasal, oral, and laryngeal warts.

HPV types 6 and 11 rarely are associated with invasive invasive squamous cell carcinoma of stds the external genitalia.

Depending on the size and anatomic location, genital warts can be painful, friable, and pruritic, although they are commonly asymptomatic.

HPV types 16, 18, 31, 31, 33, and 35 stds are found occasionally in visible genital warts and have been associated with external genital (i.e., vulvar, penile, and anal) squamous intraepithelial neoplasia (i.e., squamous cell carcinoma in situ, bowenoid papulosis, papulosis, Erythroplasia of Queyrat, or Bowen's disease of stds the genitalia) .

These HPV types also have been associated with vaginal, anal, and cervical intraepithelial dysplasia and squamous cell carcinoma.

Patients who have visible genital genital warts can be infected simultaneously with multiple HPV types.

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Treatment

The primary goal of treating visible genital warts is the removal of symptomatic warts.

In most patients, treatment can induce wart-free wart-free periods.

If left untreated, visible genital warts may resolve on their own, remain unchanged, or increase in size or number.

Determining whether treatment of genital warts will reduce transmission is difficult, because no stds no laboratory marker of infectivity has been established and because clinical studies evaluating stds the persistence of HPV DNA in genital tissue after treatment have shown variable results.

Existing data indicate that currently available therapies therapies for genital warts may reduce, but probably do not eradicate, infectivity.

Whether stds the reduction in viral DNA that results from current treatment regimens impacts future transmission remains unclear.

No evidence indicates that either either stds the presence of genital warts or their treatment is associated with the development of cervical cancer.

Regimens

Treatment of genital warts should be guided by the preference of the patient, the

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By Burt Lidenwald